cell death

Multicellular organisms can self-destruct their cells through a variety of mechanisms.

Low oxygen resuscitates gene transcription after death.

Digging into how and why some genes are resurrected after death sounds morbid, but it has practical applications. 

Urinary tract infections leave permanent epigenetic marks in the mouse bladder epithelium, reprogramming its response to subsequent infections, a study finds.

A study’s authors say their oxygenating cocktail may lead to technologies that preserve organs in deceased people for longer periods for transplantation.

Though scientists don’t yet know much about it, a newly described process called erebosis might have profound implications for how the gut maintains itself.

A new study reports that human colon cancer cells at imminent risk of death can instead develop characteristics needed to colonize new parts of the body.

The Salk professor developed the institute’s first neurobiology lab and used it to develop cell lines, describe amyloid β toxicity, and screen for compounds that protect against neurodegeneration.

Drugs targeting patients’ immune systems, rather than the virus itself, could be key to recovery from severe cases of the disease, some researchers suggest.

Introducing either necroptotic cells or an enzyme that triggers necroptosis can wipe out cancer.

Telomeres in cancer cells exposed to oxidative stress got shorter and stickier.

Internalizing the environment might be the next step in humans’ relationship with our planet.

Apoptosis and other types of programmed cell death appear to be reversible.

Molecular programs can rescue cells already engaged in the process of apoptosis or other forms of programmed cell death.

Researchers identify antibacterial functions of cell death in Arabidopsis when the plant is infected with Pseudomonas.  

The biologist earned a Nobel Prize in 2002 for his work on C. elegans.